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  Indian J Med Microbiol
 

Figure 1: Schematic diagram of the mechanism proposed for PRES secondary to blood transfusion. The most commonly described pathophysiological mechanisms to PRES are related to the self-regulation of cerebral blood vessels. One proposes that volume overload hypertension exceeds the cerebral vessels' self-regulation capacity. The other suggests that cerebral vasoconstriction occurs due to excessive autoregulation. IL-1: Interleukin-1, TNF: Tumor necrosis factor, PRES: Posterior reversible encephalopathy syndrome

Figure 1: Schematic diagram of the mechanism proposed for PRES secondary to blood transfusion. The most commonly described pathophysiological mechanisms to PRES are related to the self-regulation of cerebral blood vessels. One proposes that volume overload hypertension exceeds the cerebral vessels' self-regulation capacity. The other suggests that cerebral vasoconstriction occurs due to excessive autoregulation. IL-1: Interleukin-1, TNF: Tumor necrosis factor, PRES: Posterior reversible encephalopathy syndrome